thra gene

A novel thyroid hormone receptor encoded by a cDNA clone from a human testis library.

gene perturbations changing expression of THRA gene from the MSigDB Signatures of Differentially Expressed Genes for Cancer Gene Perturbations dataset. PI3K, phosphoinositide 3-kinase; MAPK/ERK, mitogen-activated protein kinase/extracellular signal-regulated kinase; NCX, Na+/Ca2 + exchanger; NOS, nitric oxide synthase. (1987) The physical map of the whole E. coli chromosome: application of a new strategy for rapid analysis and sorting of a large genomic library. The cDNA encodes a protein of 490 amino acids with a molecular mass of 54.8 kD. Visser et al. Comparing the relative amounts of the different THRs between the tissues during the life span remains a challenge, especially at the protein level. [Full Text], Sakurai, A., Nakai, A., DeGroot, L. J. about genes and proteins. science writers and biocurators. In an 8-year-old Polish girl with congenital nongoitrous hypothyroidism (CHNG6; 614450), Tylki-Szymanska et al.

Note: Erratum: New Eng. Metabolism 21: 723-756, 1972. There is no available study on the distribution and functional differences between these isoforms that probably results from alternative promoter usage. (1972) Revised structure of aspartokinase I-homoserine dehydrogenase I of Escherichia coli K12.

[Full Text], Jhanwar, S. C., Chaganti, R. S. K., Croce, C. M. Eur. Diseases associated with THRA include Hypothyroidism, Congenital, Nongoitrous, 6 and Resistance To Thyroid Hormone Due To A Mutation In Thyroid Hormone Receptor Alpha. The TR-α gene contains 10 exons, and the alternative splice site for TR-α2 is located on exon 9 (Fig. [Full Text: https://academic.oup.com/hmg/article-lookup/doi/10.1093/hmg/10.23.2701], Puzianowska-Kuznicka, M., Krystyniak, A., Madej, A., Cheng, S.-Y., Nauman, J. Meta-analyses of genes modulating intracellular T3 bio-availability reveal a possible role for the DIO3 gene in osteoarthritis susceptibility. A more complex ontogenic pattern was observed for TR-beta-1, suggestive of a nadir between 8.4 and 12.0 weeks' gestation. source=DH10B Chew, in Encyclopedia of Neuroscience, 2009.
CHNG6,

The THRA gene (also known as c-erbA-alpha), located on chromosome 17, has one product, THRα1, that is able to bind T3 and another product, α2, that is incapable of binding T3.69 However, the THRB gene, located on chromosome 3, expresses two T3-binding isoforms, TRβ1 and THRβ2 (THRβ3 is found only in rats),70 that mainly differ in their tissue distribution and in the length of the A/B domain.71 All THR isoforms contain highly homologous DBD, LBD, and transactivation domains (Fig.

[Isoform Alpha-2]: Does not bind thyroid hormone T3. mRNA differential expression in normal tissues and medical or genetic condition are urged to consult with a qualified physician for diagnosis and for answers to personal questions.
Vincent Laudet, Hinrich Gronemeyer, in The Nuclear Receptor FactsBook, 2002. [Full Text]. An additional truncated species was detected with the TR-alpha-2 primer set, consistent with the TR-alpha-3 splice variant described in rat. K-12 substr. Spaulding, in Reference Module in Biomedical Sciences, 2014. [Full Text], Miyajima, N., Horiuchi, R., Shibuya, Y., Fukushige, S., Matsubara, K., Toyoshima, K., Yamamoto, T. The mechanisms underlying this regulatory effect have been studied first by showing in vitro that Rev-erbα sequences regulate TRα splicing55. Constitutional t(15;17).

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